Repository logo
  • English
  • 中文
  • Log In
    New user? Click here to register.Have you forgotten your password?
Repository logo
    Communities & Collections
    Research Outputs
    Fundings & Projects
    People
    Organizations
    Statistics
  • English
  • 中文
  • Log In
    New user? Click here to register.Have you forgotten your password?
  1. Home
  2. .TMU Publications / 北醫出版品(教師升等著作 / 教學實踐 / 學位論文)
  3. .博碩士學位論文
  4. 107學年度
  5. 臺灣特有種植物成分及其改善 H2O2 誘導 BV-2 小神經膠細胞衰老功效研究
 
  • Details
Options

臺灣特有種植物成分及其改善 H2O2 誘導 BV-2 小神經膠細胞衰老功效研究

Other Title
The Constituents from Taiwanese Endemic Plant and Their Effect on Attenuating H2O2-induced Senescence in BV-2 Microglia
Type
thesis
Date Issued
2019-07-11
Author(s)
張悅行
Advisor
李美賢
Subjects
系所名稱:生藥學研究所
Description
學位別:碩士
語文別:中文
指導教授:李美賢
口試委員:沈郁強 Yuh-Chiang Shen;蕭哲志 George Hsiao
電子論文連結:https://handle.ncl.edu.tw/11296/zdpq43
URI
https://203.71.86.71/handle/123456789/60600

Copyright Notice

● The digital content on this platform is part of the Taipei Medical University Institutional Repository, featuring various academic works and outputs from the institution. It offers free access to academic research and public education for non-commercial use.

● Please use the content appropriately and within legal boundaries to respect copyright owners' rights. For commercial use, please obtain prior authorization from the copyright owner. Users must not use TMUIR for any illegal purposes.

● By utilising the platform, users are deemed to have fully accepted and understood all the regulations set out in this statement, relevant laws of the Republic of China, all international internet regulations, and usage conventions.

● TMUIR is committed to protecting the interests of copyright owners. If you believe that any material on this website infringes copyright, please contact our staff at libirtmu@gmail.com, and we will remove the work from the repository.

Built with DSpace-CRIS software - Extension maintained and optimized by 4Science

  • Cookie settings
  • Privacy policy
  • End User Agreement
  • Send Feedback