Repository logo
  • English
  • 中文
  • Log In
    New user? Click here to register.Have you forgotten your password?
Repository logo
    Communities & Collections
    Research Outputs
    Fundings & Projects
    People
    Organizations
    Statistics
  • English
  • 中文
  • Log In
    New user? Click here to register.Have you forgotten your password?
  1. Home
  2. College of Nutrition / 營養學院
  3. School of Nutrition and Health Sciences / 保健營養學系
  4. 砷誘發活性氧物種對血管致粥瘤性機轉及基因多形性之探討---砷化合物對囓齒類動物與肝細胞株氧化機制之探討(I)
 
  • Details
Options

砷誘發活性氧物種對血管致粥瘤性機轉及基因多形性之探討---砷化合物對囓齒類動物與肝細胞株氧化機制之探討(I)

Other Title
The Oxidative Mechanism of Arsenic Compounds in Murine and Hepatic Cell (I)
Type
report
Date Issued
2003
Author(s)
黃士懿  
陳玉華  
楊素卿  
Subjects
保健營養學系
計畫報告
URI
https://203.71.86.71/handle/123456789/21878
File(s)
Loading...
Thumbnail Image
Name

attachment.pdf

Size

144.59 KB

Format

Adobe PDF

Checksum

(MD5):d249cbb35e18819dd4a3b0e4f7f541c0

Copyright Notice

● The digital content on this platform is part of the Taipei Medical University Institutional Repository, featuring various academic works and outputs from the institution. It offers free access to academic research and public education for non-commercial use.

● Please use the content appropriately and within legal boundaries to respect copyright owners' rights. For commercial use, please obtain prior authorization from the copyright owner. Users must not use TMUIR for any illegal purposes.

● By utilising the platform, users are deemed to have fully accepted and understood all the regulations set out in this statement, relevant laws of the Republic of China, all international internet regulations, and usage conventions.

● TMUIR is committed to protecting the interests of copyright owners. If you believe that any material on this website infringes copyright, please contact our staff at libirtmu@gmail.com, and we will remove the work from the repository.

Built with DSpace-CRIS software - Extension maintained and optimized by 4Science

  • Cookie settings
  • Privacy policy
  • End User Agreement
  • Send Feedback